Myocardial Metabolism in Reactive Hyperemia
نویسندگان
چکیده
منابع مشابه
Adenosine metabolism in canine myocardial reactive hyperemia.
In pentobrabital-anesthetized open chest dogs, myocardial adenosine content is elevated by 5 or 15 seconds of left coronary artery occlusion and falls exponentially to control levels during reactive hyperemia. The rate constants for adenosine dissipation are (mean +/- SEM): -0.08 +/- 0.01 and -0.034 +/- 0.007 sec-1 after 5- and 15-second occlusion, respectively. Kinetic analysis of the reactive...
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OLSON, RAY A., AND DONALD E. GREGG Myocardial reactive hyperemia in the unanesthetised dog. Am. J. Physiol 208(2): 224230. I g65.-Studies of myocardial reactive hyperemia in dogs 5-24 days after the implantation of electromagnetic flowmeters on coronary artery branches reveal that the duration of occlusion and the control flow rate are independent determinants of the volume of reactive hyperemi...
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one subject with isolated, fixed coronary obstruction. Three subjects with variant angina had short episodes of ischemic ST-segment elevation that lasted 20-100 seconds. Thallium scintigrams demonstrated excess uptake in regions judged to be ischemic by angiographic and electrocardiographic criteria. Two subjects, one with variant angina and the other with a fixed coronary lesion, had prolonged...
متن کاملContribution of anaerobic metabolism to reactive hyperemia in skeletal muscle.
Elevated blood flow (reactive hyperemia) is seen in many organs after a period of blood flow stoppage. This hyperemia is often considered to be due in part to a shift to anaerobic metabolism during tissue hypoxia. The aim of our study was to test this hypothesis in skeletal muscle. For this purpose we measured NADH fluorescence at localized tissue areas in cat sartorius muscle during and after ...
متن کاملReduction of myocardial reactive hyperemia during oxygen breathing in dogs.
In 7 open-chest anesthetized mongrel dogs the left anterior descending coronary artery was occluded for 20 sec. Myocardial reactive hyperemia was recorded during air and oxygen breathings. The excess blood flow during the reactive hyperemia was smaller and the duration of the reactive hyperemia was shorter during oxygen breathing than during air breathing. It is suggested that the reduction of ...
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ژورنال
عنوان ژورنال: Japanese Circulation Journal
سال: 1970
ISSN: 0047-1828,1347-4839
DOI: 10.1253/jcj.34.413